Friday, November 4, 2011


The patient often will report a sudden onset of either a single or multiple floating spots, along with flashing lights (photopsia). Visual symptoms will be stable within the patient's visual field. There may be precipitating ocular or head trauma. If there is a posterior vitreous detachment, there will also be one large floater. If there has been a vitreous hemorrhage, there will be multiple floaters. There may be a severe loss of vision if there is a dense vitreous hemorrhage or rhegmatogenous retinal detachment. However, in a number of cases, the patient is either asymptomatic or experienced symptoms so long ago that they were forgotten.

Retinal tears result from the vitreous pulling free from the retina during vitreous detachment. During the course of the PVD, the vitreous may encounter an area where it is firmly attached to the retina. These include the optic disc, macula, along blood vessels, vitreous base, at areas of chorioretinal scarring, along the edges of lattice degeneration, and at vitreoretinal tufts. Traction at any one of these points may result in the vitreous pulling the retina free from its loose attachment to the retinal pigment epithelium with a tear developing within the sensory retina. The subsequent break in the retina can allow liquid vitreous to enter the potential subretinal space, resulting in a rhegmatogenous retinal detachment. If the vitreous remains attached to the damaged retina, traction on the edges of the break can serve to further separate the retina from the RPE.

There are three types of tractional retinal tears: the flap tear, the tear along lattice lesions, and an operculated tear. In the classic flap (or horseshoe) tear, the retina is pulled incompletely free and forms a triangular appearance. The apex of the tear is still attached to the mobile vitreous and points towards the posterior pole. The base of the triangle parallels the vitreous base. The mobile vitreous acts to further tear the retina and separate it from the RPE. If the tear bridges a blood vessel, there can be a subsequent vitreous hemorrhage. A similar retinal tear occurs at the posterior border of lattice lesions due to the same forces, but does not have the triangular appearance of the flap tear. If an area of retinal tissue is pulled completely free by the vitreous, it is considered an operculated tear. The retinal tissue, now termed an operculum, is seen to float in the vitreous above the retinal tear.

In any case of retinal tear, if the vitreous is still attached and exerting traction on the retina through the break, the mechanical forces on the retina will be perceived by the patient as flashing lights. This indicates that there are forces active on the break which may lead to further separation of the retina from the RPE. As liquid vitreous gains access to the subretinal space, the retina is further separated from the RPE, and a rhegmatogenous retinal detachment can form.

The standard management of tractional tears has always been prophylactic laser photocoagulation or cryoretinopexy. This creates an RPE hyperplastic scar around the break and seals the retina to the RPE, thus preventing the accumulation of subretinal fluid and subsequent rhegmatogenous retinal detachment. However, not all cases benefit from prophylactic treatment. If the patient is aphakic or pseudophakic, has a history of previous retinal detachment in either eye, is about to undergo ocular surgery, or if the tear is fresh or associated with any hemorrhage, then the patient should receive prophylactic therapy.

If the patient is symptomatic with photopsia, or if there is more than one disc diameter of subretinal fluid or elevation extending beyond the edge of the break, the patient needs treatment, as the risk of detachment is high. Any tractional tears along the edge of lattice lesions also require treatment. If there are none of the above risk factors, the patient is asymptomatic and there is no subretinal fluid, monitor the patient on a six-week, three-month, six-month, 12-month schedule. If you see progression at any follow up visit, have the patient receive prophylactic treatment.


The greater the length of time a tractional tear, or any retinal break, exists in an untreated eye without progressing to retinal detachment, the less likely the chance that it will progress.
Most tractional tears without symptoms or risk factors can be safely monitored without treatment. Often, the RPE will become hyperplastic due to the insult from the tear and form a chorioretinal scar around the break. If this happens, it becomes very unlikely that the tear will ever lead to detachment.
Retinal breaks located superiorly in the retina are no more likely to progress to retinal detachment than are breaks located inferiorly in the retina. Location of the break should not be considered when determining risk of detachment.

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